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Source: Vitality Connect GAINSWave

Unfortunately, many men are reluctant to report erectile dysfunction (ED) to their partner, or physician, due to social stigmas associated with bedroom performance. ED, or the inability to maintain an erection, is much more common than the general population may realize and has a lot to do with men’s health. A Johns Hopkins study revealed that over 18 million men in the U.S. over the age of 20 experience difficulties having and maintaining an erection.

Signs & Symptoms:

Erectile Dysfunction (ED), also known as impotence, is a type of male sexual dysfunction characterized by the inability to maintain an erection long enough and firm enough for sexual intercourse.

While most men will have occasional difficulty achieving a healthy erection during sexual intercourse, ED is only considered a possible medical explanation for these symptoms if erection difficulties have been affecting a man for an extended period of time. When blood flow to the two chambers of the penis, known as the corpora cavernosa, are blocked or restricted for any reason, this can result in ED.

Erectile Dysfunction Causes:

Men worldwide suffer from erectile dysfunction due to physical, psychological or emotional reasons. Many of which are seeking a natural, ED solution with long-term results.

What causes ED? There are many causes of impotence both medical and psychological. Often, age is a predominant factor in erectile dysfunction; however, your chronological age is not solely responsible for ED’s development. Men over the age of 40 typically experience ED as the result of inadequate blood flow to the penis, most often due to the narrowing of blood vessels. This can arise due to many causes including:

Poor circulation

The most common cause of ED is a lack of flowing blood to erectile tissue. Plaque and damaged vessels can restrict circulation enough to disrupt function. Typically, symptoms begin with difficulty maintaining an erection before advancing to a complete inability to obtain one.

Hormonal imbalances

The production of testosterone and other hormones naturally decreases with age, and this can disrupt erections. Also, in rare instances, kidney failure and liver disease may throw off the balance of hormones necessary for proper erection and intercourse. In most cases, testosterone replacement therapy can bring levels of the hormone back to normal.

Obesity or lack of physical activity

Engaging in an active lifestyle can help with weight management, stress, anxiety, and depression – all of which help with other conditions and decrease the risk of developing ED. The accumulation of fat in the body can cause a drop in active testosterone levels. In addition, being overweight can cause cardiovascular problems that are known to contribute to ED.

Alcohol consumption

More than two drinks a day is considered a risk factor for ED. Alcohol can lower testosterone levels, decrease flowing blood to the penis, and cause nerve and hormonal imbalances. Low testosterone from alcohol or drug abuse can also affect your libido and performance, potentially leading to further psychological ED complications.

Smoking

The hardening of the arteries associated with smoking tobacco can reduce flowing blood to the penis. Smoking also results in oxidative stress – another risk factor for erectile dysfunction. Fortunately, quitting smoking has shown to relieve much of the ED risks associated with the habit.

Cycling and horseback riding

The pressure applied to the pelvic floor during these activities can result in nerve and vascular damage that may inhibit erections.

Sleep apnea

A German study found that 69% of men involved in a sleep apnea study also suffered from ED. Disruptions to breathing during sleep affect oxygen levels in the blood, which can cause problems generating an erection. Lack of sleep also disrupts hormone production.

Psychological difficulties

Stress, both in and out of the bedroom, can make it difficult for a man to achieve an erection. Depression, anxiety, and the medications to treat them may also contribute to impotence.

Poor dental health

A recent study published in the Journal of Sexual Medicine reported that men with severe periodontal disease were three times more likely to have consistent difficulties having an erection. While this may be a case of correlation rather than causation, poor dental health has long been associated with vascular disease, which is known to contribute to ED.

Peyronie’s disease

This condition is characterized by the bending or curvature of the penis, or narrowing of the penile shaft. Scarring related to Peyronie’s disease can cause circulation problems in the penis.

High cholesterol

Cholesterol can cause damage to blood vessel linings in all parts of the body – including the penis. Blockages caused by elevated cholesterol levels or plaques that restrict flowing blood can prevent erectile tissue from filling with blood. A healthy diet and lifestyle, including regular exercise, can help control these levels and the risk of developing ED. In addition, doctors may prescribe pharmaceuticals to lower cholesterol in the blood.

Metabolic syndrome (MetS)

MetS is a combination of maladies that can cause ED. The five hallmarks of MetS are: hypertension, elevated fasting glucose levels, elevated triglycerides, central obesity, and reduced high density lipoprotein levels of cholesterol. MetS causes inflammation and hormonal imbalances that can lead to ED. Treatment for MetS can include lifestyle changes, testosterone replacement, pharmaceuticals, and bariatric surgery – such as gastric bypass.

Hypertension

30-50% of hypertensive men also suffer from ED. Hypertension can disrupt erectile tissue function. In addition, some prescriptions for the condition can cause ED. However, there’s evidence that suggests that once adequate blood pressure control is regained – even for those on medication – ED becomes less likely.

Diabetes

35-90% of diabetic men also experience ED. Hyperglycemia can have a negative impact on several aspects of erectile function. Complications and changes associated with diabetes can compromise macrovascular and microvascular structures, the nervous system, and blood vessel linings.

A healthy diet & exercise

One study indicated that intensive glycemic control might decrease the chances of ED in diabetic men.

In response, a number of erectile dysfunction treatments have been developed, with varying efficacy and side effects.

ED Medications:

Among the most popular treatment options for erectile dysfunction are pharmaceuticals. Drugs such as Viagra (Sildenafil), Levitra (Vardenafil), and Cialis (Tadalafil) are popular and boast an 80% success rate. These drugs are classified as phosphodiesterase-5 inhibitors (PDE-5) – which enable an erection, temporarily, but are not the best for maintaining an erection over time.

Prescriptions are heavily relied upon, having to be taken 15 minutes to 36 hours before sexual activity is initiated – not only reducing the spontaneity of one’s sex life but oftentimes fostering dependence.

This route typically isn’t recommended for men taking nitrate drugs or alpha-blockers, and all medications, including over-the-counter herbs and supplements, should be shared with your doctor to prevent negative interactions. The commonly reported potential side effects of phosphodiesterase-5 inhibitors include headache, flushing, runny nose, stomach pain, back pain, indigestion, vision changes, and dizziness.

Treatment options for erectile dysfunction don’t have to be so much work.

GAINSWave Treats the Root Cause of ED:

GAINSWave®, the clinically proven, non-invasive therapy, was specifically developed for men seeking an erectile dysfunction treatment that would improve their sexual performance.

The pulsating waves of GAINSWave improves blood flow to the penis by dissolving plaque build-up and stimulating the growth of new blood vessels. The improvement results in stronger, harder, and more sustainable erections.

The pulsating waves also trigger a healing response that “wakes-up” dormant stem cells and growth factors in the penis for an overall rejuvenation of male erectile tissue.

With this alternative ED treatment, there are no expensive prescriptions to remember to take or refill, and no penile implants or surgery. There are none of the harmful side effects with this ED treatment as with oral drugs.

GAINSWave therapy is effective and safe for men who cannot take oral ED medicine, such as those with heart disease, diabetes, or high blood pressure. GAINSWave can be used to treat Peyronie’s Disease, which Viagra and other oral ED drugs cannot.

Sign up for your FREE GAINSWave Phone Consultation today!

Enhanced Wellness Living, Ridgeland, MS, is proud to offer an array of sexual wellness treatment options for both men and women!

Votiva for Her  |  O-Shot for Her  |  GAINSWave for Him  |  P-Shot for Him

GAINSWave for Him + Votiva for Her

EWLiving.Info@gmail.com

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Enhanced Wellness Living of Ridgeland, Mississippi is your leading Functional Medicine, Holistic Health, Lifestyle Medicine, and Integrative Medicine clinic!  Nurse Practitioner, Kelly Engelamann, and the Enhanced Wellness Living team have supported all of Mississippi including Ridgeland, Jackson, Madison, Brandon, Hernando, Starkville, Flowood, Vicksburg, Oxford, Olive Branch, the Mississippi Gulf, Cleveland, Clindon, Hattiesburg, Canton, Bolton, Hinds, and more since 2004!

These statements are not intended to diagnose, treat, or cure any disease.

At Enhanced Wellness Living we practice a food first approach to healing.  Nurse Practitioner, Kelly Engelmann, founded her Functional Medicine clinic through dedicating years to researching and developing treatment strategies using a holistic health approach for healing her patients.  Kelly is proud to be at the forefront of Functional Medicine and to share resources of fellow integrative medicine practitioners, like this publication by Chris Kresser. 

For more information on our approach to healing and preventing chronic disease compelte the “Get Started Today” and we will contact you.

Source: Chris Kresser 

Find out which ingredients you should be
concerned about, and which are safe
Food Additives: Harmful or Harmless?
One of the benefits of ancestral eating is that you avoid potentially harmful food additives
like artificial colors, MSG, and artificial sweeteners. But even on a Paleo diet, it can be
hard to avoid some fillers, thickeners, and additives.
In fact, many common Paleo foods contain more additives than their Neolithic
counterparts! For instance, commercial nut milks and coconut milk often contain
thickeners like gums or carrageenan, while your run-of-the-mill grocery store whole
cow’s milk is additive-free.
In this eBook, I’ll review the science on some of the most common additives, and let you
know whether you should be concerned about consuming them. First up – magnesium
stearate!

Magnesium Stearate
Magnesium stearate is a salt that is produced when a magnesium ion bonds with two
stearate molecules. Stearate is just the anion form of stearic acid, which you’ve most
likely heard of before. Stearic acid is a long-chain saturated fat that is abundant in beef,
cocoa butter, coconut oil, and other natural foods. It’s also the only long-chain saturated
fat that scientists and medical practitioners agree doesn’t raise cholesterol levels, and
doesn’t increase risk of heart disease.
Magnesium stearate is most commonly used in supplement manufacturing as a ‘flow
agent,’ which helps ensure that the equipment runs smoothly and the ingredients stay
blended together in the correct proportions.

Given the seemingly benign components of this additive, it’s a little surprising how
controversial it is. There are a lot of misconceptions and inaccurate statements about it
floating around the internet, and while I wouldn’t recommend consuming vats of the stuff
(not that you’d want to), I think the concern over magnesium stearate is largely
overblown.

EFFECT ON IMMUNE CELLS
One study that many people have used as evidence against magnesium stearate is a
1990 experiment entitled “Molecular basis for the immunosuppressive action of stearic
acid on T cells.” This baffles me, and I suspect that anyone using this study to indict
magnesium stearate hasn’t actually read it.
In the experiment, scientists isolated T-cells and B-cells from mice, put them in a Petri
dish, and bathed them in a solution containing stearic acid (along with some other
components). They observed that the T-cells incorporated the stearic acid into their cell
membrane, eventually de-stabilizing the membrane enough that the cell died.
First of all, this study has nothing to do with magnesium stearate. They just used the plain
old stearic acid that you’d find in your beef, chocolate, or coconut oil, so this study could
just as easily be used against those foods. If you’re going to be concerned about this
study (which you shouldn’t be), you’d have much bigger sources of stearic acid to worry
about than the magnesium stearate in your supplements.
Second, the study has nothing to do with stearic acid consumed in the diet. Under
normal conditions, your T-cells are not bathed in stearic acid, even if you consume
superhuman amounts of coconut oil, tallow, and cocoa butter.
Finally, the researchers used T-cells from mice, and in this case, the results cannot be
applied to humans. The mouse cells incorporated stearic acid into their membranes
because they lacked the ability to de-saturate fatty acids. However, human T-cells do
have the ability to de-saturate fatty acids, so even if you did bathe your T-cells in stearic
acid, they would be able to maintain their membrane function. (1)

In case you got lost, here’s a summary: this study has no relevance whatsoever to human
consumption of magnesium stearate, I have no idea why the study is being referenced in
this manner, and you shouldn’t be concerned about it.
CONCERNS ABOUT PESTICIDES AND GMOS
Another criticism is that because stearate is often derived from cottonseed oil, it can be
contaminated with pesticides. Keep in mind that magnesium stearate is a highly purified
substance, and goes through an intensive refining process before appearing in your
supplements. So far, I haven’t come across any reports indicating that magnesium
stearate retains substantial amounts of pesticide residue.
As for the concern that cottonseed oil is often genetically modified, the source of crude
fat shouldn’t make a difference in the final form of the stearate. Stearic acid is an 18-
carbon molecule with a specific chemical structure that will be the same whether the
stearic acid is from a genetically modified cotton plant, a bar of Hershey’s chocolate, or a
grass-fed ribeye steak.
EFFECT ON NUTRIENT AND DRUG ABSORPTION
Another criticism is that magnesium stearate might inhibit nutrient absorption. One in
vitro study conducted in 2007 found that tablets containing magnesium stearate
dissolved more slowly than tablets without magnesium stearate when placed in artificial
gastric juice. (2) The study authors concluded that in vivo studies are needed to
determine whether this finding has any practical significance. However, an earlier study
found that although magnesium stearate increased the time it took for a drug to dissolve,
it had no effect on overall bioavailability, as evidenced by blood levels of the drug in test
subjects. (3) Further, another study found that levels of magnesium stearate didn’t affect
tablet dissolution at all. (4)
All of this indicates that although magnesium stearate might affect the rate of tablet
dissolution in some circumstances, it doesn’t affect the overall bioavailability of the drug
or supplement.
BIOFILMS
I’ve seen this claim pop up in a few places around the internet, so I’ll address it briefly.
Some critics of magnesium stearate claim that it can induce formation of harmful biofilms

in the intestine. This assertion appears to be based on the fact that soap scum contains
magnesium and calcium stearate, so they insist that just as soap scum creates films on
your sink or shower, magnesium stearate creates films on your intestines.
It should be pretty obvious that the intestinal lumen is a vastly different environment from
a shower door, but some people still seem to be concerned. Rest assured, there is no
conceivable reason why this would take place, and I haven’t seen a single scientific
article that even hints at this possibility.
ALLERGIES
A 2012 study entitled “Magnesium stearate: an underestimated allergen” reported on
a 28-year-old woman who had an allergic reaction to magnesium stearate, resulting in
hives. I’m very curious about this, because an allergy to either magnesium or stearate
seems highly unlikely, but unfortunately I don’t have full-text access to that study. But
needless to say, if you develop hives (or another allergic response) after consuming
magnesium stearate, you should probably avoid it in the future.
As a final note, a rat study determined that you’d have to take 2500mg of magnesium
stearate per kilogram of bodyweight per day to start seeing toxic effects. (5) That means
a 150lb person would have to consume 170,000 mg per day, which is so far beyond any
amount you would encounter in supplements that it’s a non-issue.
Overall, I haven’t found scientific evidence to substantiate the claims against magnesium
stearate, and the small amounts found in supplements shouldn’t be a problem for the
majority of the population.

Soy Lecithin
Soy lecithin is one of the most ubiquitous additives in our food supply. It’s used primarily
as an emulsifier, and you can find it in everything from salad dressing to tea bags. Paleo
dieters avoid the brunt of it by eliminating most processed foods, but it almost always
pops up in chocolate (everyone’s favorite honorary Paleo food) and often appears in
supplements.
I recommend avoiding soy as a general rule, but consuming small amounts of soy
lecithin as an additive is very different from, say, eating a soy burger topped with soy
cheese or drizzling soybean oil on your salad.
WHAT IS SOY LECITHIN?
The term ‘lecithin’ can have different meanings depending on the context, but for our
purposes, it refers to a mixture of phospholipids and oil. Phospholipids are a component
of the cell membrane in all plants and animals, but lecithin is most often derived from
sunflower kernels, rapeseed (canola), milk, soy, and egg yolks. (6)
The specific composition of soy lecithin varies depending on its manufacturer and
intended use, but on average, it contains about 35% soybean oil and 16%
phosphatidylcholine. (7) Phosphatidylcholine is a type of phospholipid that is abundant in
liver and egg yolks, and is the primary form of choline found in foods. (8) The remaining
percentage is other phospholipids and glycolipids.
To make soy lecithin, soybean oil is extracted from the raw soybeans using a chemical
solvent (usually hexane). (9) Then, the crude soy oil goes through a ‘degumming’
process, wherein water is mixed thoroughly with the soy oil until the lecithin becomes
hydrated and separates from the oil. Then, the lecithin is dried and occasionally bleached
using hydrogen peroxide.
There are many claims online about soy lecithin being full of nasty chemicals left over
from the production process. Not surprisingly, there aren’t many credible sources
describing the chemical content of commercial soy lecithin, but I have found some
relevant data about the safety of soy lecithin.
Before the ‘degumming’ step where lecithin is removed, the crude oil undergoes a multistep process to remove the hexane. (10) However, it appears that the FDA doesn’t
regulate the amount of hexane residue in food products, and one paper estimated that
the residual hexane concentration of soy oil is 500-1000ppm. (11) So, it’s very possible
that similar concentrations remain in the soy lecithin. (For comparison’s sake, the
concentration limit for hexane in pharmaceuticals is 290ppm.) (12)
According to one analysis, total pesticide residues in crude soy oil are around 400ppb.
(13) Since the pesticide concentration of the oil after degumming is similar, it’s pretty
likely that some of those pesticides end up in the lecithin as well.
While it’s unfortunate that soy lecithin likely contains pesticides and solvents, I would just
encourage you to keep this information in perspective. We’re exposed to hundreds of
chemical toxins every day in our air, water, household products, and food, and
contaminants in soy lecithin will contribute only slightly to your overall toxic load. After all,
we’re talking parts per million and parts per billion, and soy lecithin itself usually makes
up no more than 1% of processed foods. (14)
Of course, in an ideal world, we would be able to avoid these things altogether, and I
certainly recommend reducing your exposure as much as possible. It’s also a good idea
to make sure your detox systems are functioning effectively. But unless you have a
severe chemical sensitivity to hexane or pesticides, occasionally consuming small
amounts is not worth getting bent out of shape over.
ALLERGIES
Soy allergies are triggered by soy proteins, so whether lecithin triggers an allergic
response or not depends on its protein content. One analysis found protein
concentrations ranging from 100 to 1,400ppm in six different soy lecithin samples. (15)
(For reference, the new FDA gluten-free labeling law requires a gluten concentration of
less than 20ppm.) (16)
Another analysis of six different lecithin samples found that four had sufficient protein to
trigger an IgE-mediated response in people with soy allergies, while two contained no
detectable protein at all. (17) However, another study performed similar testing and
concluded that even if protein is present in soy lecithin, it’s not a significant allergen for
people with soybean allergies. (18)
It’s clear that the source of the soy lecithin is a major determinant in whether or not it will
present a problem for those with soy allergies, but if you have a soy allergy, I’d say better
safe than sorry. However, because protein is present in such a low concentration, and
soy lecithin itself usually makes up no more than 1% of processed foods, it’s probably not
a problem for those with minor sensitivities to soy.
GMO
Most of the soy grown in the US is genetically modified, so unless the label says ‘organic
soy lecithin,’ it probably came from a genetically modified soybean. You know I’m not a
fan of GMOs, due to the presence of potentially transferrable DNA and potentially
immunogenic proteins.
However, as I discussed in the section on allergies, soy lecithin contains very little soy
protein, and lecithin from some sources contains no detectable protein at all. Soy lecithin
also contains very little DNA, and the DNA present is usually degraded to the extent that
it’s impossible to tell whether the soy is genetically modified or not. (19) Thus, most of the
risks associated with consumption of GMOs aren’t relevant for soy lecithin, and shouldn’t
be cause for concern.
PHYTOESTROGENS
Soy is the greatest food source of phytoestrogens, and one group of researchers
discovered significant estrogenic activity in soy lecithin. (20) Interestingly, none of the
soy lecithin they tested contained genistein, which is the predominant phytoestrogen in
soy. They concluded that “a so-far unidentified estrogen-like compound” is present in
soy lecithin that accounts for its estrogenic activity.
We know how problematic phytoestrogens can be, but again, the dose makes the
poison. Remember, soy isn’t the only source of phytoestrogens we’re exposed to. (Did
you know that flaxseed is also a significant source of phytoestrogens? In fact, one study
showed that supplementation with ground flaxseed altered estrogen metabolism even
more than supplementation with soy flour.)
It’s definitely best to keep phytoestrogens to a minimum, and individuals dealing with
cancer or fertility problems might want to avoid them more strictly. But for most generally
healthy people, the small amounts of phytoestrogens from soy lecithin shouldn’t be a
problem.
TOXICITY
One study that has been used widely as ammunition against soy lecithin is titled “Effects
of a Commercial Soy Lecithin Preparation on Development of Sensorimotor Behavior
and Brain Biochemistry in the Rat.” Researchers found that soy lecithin in
concentrations of 2% and 5% in the diets of pregnant and newborn rats resulted in
impaired reflexes and swimming ability, along with other cognitive deficiencies.
It’s important to understand that these effects are due to choline toxicity, not soy lecithin
per se. The elevated brain/body weight ratios, plus elevated acetylcholine and choline
acetyltransferase levels that resulted from soy lecithin supplementation were caused by
the phosphatidylcholine, and would’ve still occurred even if they had used a source of
phosphatidylcholine other than soy; even egg yolks.
It would be very difficult to consume as much choline as these rats did, especially from
soy lecithin. In fact, most people are deficient in choline! This is just another case of a
study being misinterpreted, and you certainly don’t need to worry about soy lecithin
causing developmental problems.
THERAPEUTIC USES
It’s worth mentioning that soy lecithin is also being recommended and consumed as a
dietary supplement. There is a growing body of research supporting its use for improving
blood lipids, reducing inflammation, and treating neurological disorders. (21) For instance,
one study found that after 2 months of supplementing with 500mg of soy lecithin per
day, total cholesterol levels fell by 42% and LDL levels decreased by 56%. (22)
However, most of these studies involve supplementation with a purified form of soy
lecithin, which usually contains less soy oil and more phosphatidylcholine than the
commercial soy lecithin that shows up in foods. Additionally, isolated phosphatidylcholine
is often referred to as ‘lecithin’ in scientific contexts, so some studies supplementing with
‘soy lecithin’ are really just supplementing with phosphatidylcholine.
So once again, it’s not the soy lecithin; it’s the choline. Luckily, you can derive all the
benefits of phosphatidylcholine supplementation just by increasing your consumption of
choline-rich foods like egg yolks and liver.
The only people who need to make a point of avoiding soy lecithin are those with severe
soy allergies or chemical sensitivities, and of course, those who notice that they
personally react badly to it. And if you don’t have a soy allergy, almost all of the
remaining concerns about soy lecithin (pesticides, solvents, and GMOs) can be
completely eliminated by purchasing products that contain organic soy lecithin.
But for the vast majority of the population, even conventional soy lecithin isn’t worth
worrying about one way or the other. If it’s just as easy for you to avoid it as it is to
consume it, then do so. (For example, Enjoy Life is one popular brand of chocolate that is
soy-free.) Ultimately, I think most people can just enjoy their occasional chocolate treat
without worrying about whether it contains soy lecithin.

Carrageenan
Carrageenan, a heavily discussed additive in the world of alternative health, is an
indigestible polysaccharide that is extracted from red algae, and is most commonly used
in food as a thickener or stabilizer. Carrageenan-containing seaweeds have been used
for centuries in food preparations for their gelling properties, but the refined, isolated
carrageenan found in modern processed foods has raised concerns in the healthconscious online community. (23)
Carrageenan is especially common in non-dairy milks such as almond milk and coconut
milk, which means that some people who transition to a Paleo diet might actually be
increasing their exposure if they use these products. I discussed carrageenan on a
recent podcast, but I want to give you a more detailed summary of the evidence.
There are a few distinct types of carrageenan that differ in their chemical properties, but
the most important distinction is between degraded carrageenan and undegraded
carrageenan. From a chemical standpoint, the difference between these two types is in
their molecular weight. From a practical standpoint, undegraded carrageenan is
approved for use in food products, while degraded carrageenan is not. (24)
Although both substances are often referred to as ‘carrageenan,’ they have very different
chemical properties and should really be treated as separate compounds. Degraded
carrageenan is also called ‘poligeenan,’ which is how I will refer to it in the rest of this
eBook to avoid any confusion.
ANIMAL STUDIES
Most of the carrageenan hysteria stems from animal studies that implicate carrageenan in
the formation of ulcerations and cancerous lesions in the colon. A thorough review of the
approximately 45 available animal studies on carrageenan was published in 2001, and at
first glance, these studies seem alarming.
However, it turns out that the majority of these animal experiments used poligeenan
instead of carrageenan, and as I mentioned before, these are two separate compounds
with different effects. Poligeenan is significantly more detrimental to the health of lab
animals than carrageenan, so the lack of a clear designation between them has given
carrageenan a worse reputation than it deserves.
One important difference is that while poligeenan can cause cancer on its own when
given in high enough concentrations, undegraded carrageenan has only ever been
shown to accelerate cancer formation when administered with a known carcinogen. (25)
In other words, food-grade carrageenan has not been shown to cause cancer in animal
models. That doesn’t necessarily mean carrageenan is in the clear when it comes to
cancer, but contrary to popular belief, it is not a known carcinogen.
Additionally, poligeenan produces more severe ulceration and inflammation than
carrageenan, and at lower concentrations. As an example, a study on rhesus monkeys
using poligeenan at 0.5-2% resulted in diarrhea, hemorrhage, and ulcerations, while
carrageenan at 1-3% resulted in no colonic changes. (26) (For reference, the
concentration of carrageenan in processed food is usually between 0.01% and 1%.) (27)
However, carrageenan has produced intestinal damage in some animal studies.
Observed effects in rats include epithelial cell loss, increased intestinal permeability, and
diarrhea. (28) In guinea pigs, carrageenan at a 5% concentration in the diet caused ulcers
in the colon, although a similar concentration in the diets of rats and hamsters resulted in
no difference from controls. (29)
In pigs, concentrations of carrageenan between .05 and .5% administered for 83 days
resulted in abnormalities in the intestinal lining, but no ulcerations or tumors. (30) Still, a
more recent rat study found no ulcerations or lesions in the colon after 90 days of
carrageenan administration. (31) These studies suggest that the effects of carrageenan
are highly species-dependent, which makes it more difficult to extrapolate these results
to humans.
There are a few other important considerations when determining how applicable these
results are to humans. Many of these experiments administered the carrageenan through
the animals’ drinking water as opposed to their food, which tends to increase the severity
of the resulting symptoms. Because carrageenan interacts with protein molecules,
consuming it as part of a solid food is much less harmful than consuming it in water.
Also, although many of the concentrations administered are comparable to
concentrations found in processed foods, many experiments were conducted at
concentrations much higher than humans would ever encounter on a normal diet.
Remember, these studies are looking at carrageenan as a percentage of the entire diet,
not just less than 1% of a small portion of the total diet, as is the case when using milk
replacement products.
HUMAN STUDIES
Experimental evidence on the effects of carrageenan in humans is extremely limited, for
obvious ethical reasons. However, a few in vitro experiments have been conducted on
isolated human intestinal cells.
One study found that in intestinal epithelial tissue, carrageenan exposure increased the
expression of two pro-inflammatory transcription factors. (32) This reaction appears to be
protective of the intestinal tight junctions, because suppression of either of the
inflammatory factors resulted in increased permeability of the isolated epithelial tissue.
Unfortunately, it’s unclear whether they used food-grade carrageenan rather than
poligeenan in this experiment.
Two similar studies that did use food-grade carrageenan also found that isolated
intestinal epithelial tissue responded to carrageenan by up regulating inflammation. (33,
34) Another study on human intestinal epithelium found that undegraded carrageenan
reduced the activity of many sulfatase enzymes, with potential negative ramifications for
the function and vitality of the cell. (35)
Finally, another study found that exposing human intestinal epithelial cells to undegraded
carrageenan in concentrations lower than what would be found in a typical diet caused
increased cell death, reduced cell proliferation, and cell cycle arrest. (36)
These studies provide some support for the generalization of the animal studies to
humans, implicating carrageenan in the potential for intestinal inflammation. However, it’s
important to remember that not only were these studies in vitro (aka not in the human
body), they also didn’t administer the carrageenan with any food, so the effects observed
may differ significantly from what actually occurs when humans ingest carrageenan in a
real-world setting.
EXPOSURE TO POLIGEENAN
Because poligeenan can be produced from carrageenan, many researchers and
laypeople have expressed concern that we might be exposed to poligeenan through
contamination of the food supply. However, the most recent sources indicate that the
poligeenan contamination level of food-grade carrageenan is less than 5%. (37)
Another encouraging data point in this situation is that while carrageenan is an extremely
effective thickener and emulsifier at concentrations as low as .01%, poligeenan has no
functional effect in food even at concentrations up to 10%. (38) Specific chemical
processing is necessary for carrageenan to be degraded to poligeenan, and because
poligeenan is of no use in the food industry, it seems unlikely that poligeenan would
show up in appreciable quantities in processed foods.
Another concern is whether small percentages of ingested carrageenan are degraded to
poligeenan in the digestive tract after consumption, either because of the acidic
environment or because of intestinal bacteria. Some experimental evidence indicates
that as much as 10-20% of carrageenan could be degraded to poligeenan during
digestion, while other researchers (not surprisingly funded by the carrageenan industry)
assert that carrageenan is stable throughout digestion. (39, 40)
Regardless, the significant differences between poligeenan and carrageenan as
evidenced by the reactions of lab animals make it pretty clear that even if some
degradation does take place, carrageenan still doesn’t have the potential for harm that
poligeenan does.
As with magnesium stearate and soy lecithin, carrageenan has been frequently portrayed
as significantly more harmful than is supported by available evidence. Contrary to
popular belief, it’s not a known carcinogen, and although some studies implicate
carrageenan in ulceration and inflammation, some show no adverse effects.
However, I do still think caution is warranted. If I had to rank additives, I’d say
carrageenan is a bit more concerning than the other two additives we’ve discussed so far
because of its association with gut issues. Remember, in cases involving modern
ingredients, the burden of proof should be on manufacturers to prove that they’re safe,
rather than on consumers to prove that they’re harmful. Because the evidence isn’t
conclusive either way, I recommend avoiding carrageenan, especially if you have a
history of digestive problems.
Personally, I adhere to the “precautionary principle” for anything I eat; in other words, in
the absence of proven safety, I choose to avoid foods that have questionable adverse
effects. Carrageenan fits this description, as there’s still some doubt about its safety and
no evidence has convinced me that there isn’t a potential for harm if consumed regularly.
Occasional exposure is likely nothing to worry about, but for most people reading this
eBook, avoiding carrageenan is probably as simple as making your own nut milk or
coconut milk, so I would encourage you to give that a shot. Also, if you follow the links to
those two posts, some commenters have shared brands of almond and coconut milk that
don’t contain carrageenan (although watch out for other additives that may be present).

Xanthan Gum
Food additives are commonly used as stabilizers, thickeners, or emulsifiers. Another
additive that shares many of these functions in commercial foods is xanthan gum, which
is also popular in gluten-free baked goods for the elasticity it lends to dough.
Although it isn’t as heavily discussed in the blogosphere as the other additives I’ve
covered thus far, many health-conscious people see it on ingredient lists and wonder
what it is, and whether they should be eating it. So now, I’ll do my best to answer those
questions.
Xanthan gum is a largely indigestible polysaccharide that is produced by bacteria called
Xanthomonas Camestris. (41) Manufacturers place the bacteria in a growth medium that
contains sugars and other nutrients, and the resulting product of bacterial fermentation is
purified, dried, powdered, and sold as xanthan gum. (Makes you wonder who first
thought to put it in food, doesn’t it?)
ANIMAL STUDIES
Overall, the results from animal studies on xanthan gum aren’t very concerning. In one
experiment, rats were fed xanthan gum for two years in concentrations of 0.25, 0.50 or
1.0 g/kg body weight per day. (42) The only notable difference between the xanthan gum
groups and the control group was that rats fed xanthan gum experienced soft stools
somewhat more frequently than the control rats, but even that barely reached statistical
significance. There were no differences in growth rate, survival, blood markers, organ
weights or tumor incidence.
Another experiment followed a similar design but used dogs instead of rats, and the
results were the same: no changes other than occasional soft stools. (43) In a threegeneration reproductive study, rats were fed either 0.25 or 0.50 g/kg per day, and there
were no significant changes in the parents and offspring from the xanthan gum-receiving
groups. (44)
Based on those initial studies, it was concluded that xanthan gum is a perfectly safe food
additive. Since then, a few additional animal studies with different aims have been
published.
One study, conducted to evaluate the effects of xanthan gum on digestion in rats, found
that a diet containing 4% xanthan gum increased the amount of water in the intestines by
400%, and also increased the number of sugars remaining in the intestine. (45) Another
study found that in rats fed 50 g/kg of xanthan gum (an incredibly high dose) for 4 weeks,
the stool water content and short-chain fatty acid (SCFA) content increased significantly.
(46)
This last study actually relates to the potential anti-tumor properties of xanthan gum, and
researchers found that orally administered xanthan gum was able to slow tumor growth
and prolong the survival of mice with melanoma. (47) The mechanism is unclear, but it’s
interesting nonetheless.
HUMAN STUDIES
Due to the lack of harmful effects observed in animal studies, there are few human
studies on xanthan gum. The first study aimed to determine the safety of xanthan gum
when consumed by humans in an everyday dietary setting, but at levels much higher
than people would normally encounter in their diet. (48) For 23 days, 5 adult men with no
GI issues consumed between 10.4g and 12.9g of xanthan gum daily (based on the
subjects’ weight), which is 15 times the current Acceptable Daily Intake of 10mg/kg.
Overall, they experienced a reduction in serum cholesterol, an increase in fecal bile acid,
and an increase in stool output and water content.
Another study had volunteers consume 15g of xanthan gum per day for 10 days. (49)
They found xanthan gum to be a “highly efficient laxative,” and subjects experienced
greater stool output and gas. That’s not very surprising considering the high dose, but
what I found particularly interesting about this study was their measurement of the ability
of subjects’ fecal bacteria to metabolize xanthan gum.
The researchers found that before the trial period, bacteria from the stools of only 12 of
the 18 subjects could break down the xanthan gum, while after the trial period, bacteria
from 16 of the subjects could break it down. (50) Additionally, the stool samples
containing bacteria that could break down the xanthan gum showed a much greater
production of hydrogen gas and SCFA after the trial period as compared to baseline,
indicating that the intestinal bacteria of the subjects quickly adapted to this new food
source. Clearly, xanthan gum (like many indigestible carbohydrates) can have a profound
impact on the gut microbiota in large doses.
COLITIS IN INFANTS
The only concerning research I found on xanthan gum relates to the development of
necrotizing enterocolitis (NEC) in infants. Recently, the New York Times published an
article relating the tragic deaths of infants who had developed NEC after consuming a
diet of formula or breast milk that had been thickened with a xanthan gum-based product
called SimplyThick. This product was widely used in hospitals to thicken feed for infants
with swallowing difficulties.
Two papers reviewed the cases of xanthan gum-associated NEC, and while there isn’t
enough data to establish causation, the general consensus seems to be that the xanthan
gum caused increased bacterial production of SCFA in the newborns’ intestines, and this
contributed to the development of NEC. (51, 52) Although SCFA are vital to colon health,
the immature digestive systems of newborns appear to be extremely sensitive to them.
(53, 54) Since then, general practice guidelines suggest avoiding manufactured
thickening products in babies under 12 months old, and rice cereal or baby oatmeal is
used instead.
I wanted to address this because while it’s clearly important to avoid giving xanthan gum
to infants (especially in large amounts), I’d like to emphasize that none of this changes
the fact that xanthan gum appears to be relatively harmless in adult humans. None of the
animal or human studies found damage to the intestinal mucosa following xanthan gum
consumption, even in large doses, so this danger appears to be unique to newborns. For
everyone else, SCFA aren’t something to be afraid of, and they are actually beneficial for
the gut and for metabolic health, as I mention in previous articles here and here.
WHEAT, CORN, SOY, AND DAIRY ALLERGIES
I mentioned in the opening section that xanthan gum is produced by bacterial
fermentation of a sugar-containing medium. Unfortunately, that ‘medium’ is often a
potentially allergenic substance such as corn, soy, dairy, or wheat. Many xanthan gum
manufacturers aren’t eager to share what their ‘medium’ is, but one common supplier,
Bob’s Red Mill, discloses their production practices.
It looks like they originally used corn or soy as a medium, but they’ve since changed
their medium to a glucose solution derived from wheat starch. However, they claim that
the xanthan gum is still gluten-free, and it continues to be marketed as such.
It can be difficult to find production info online, but just be aware that if you have a
severe allergy to corn, soy, wheat, or dairy, it would be prudent to either avoid xanthan
gum entirely or check with the manufacturer to see how it’s produced.
Based on the available evidence, the worst xanthan gum seems to be capable of (in
adults) is causing some digestive distress in those who are susceptible by increasing
stool bulk, water content, and sugar content. But as I just mentioned, those with severe
allergies should also be careful.
I recommend that people with digestive problems generally avoid xanthan gum, not
because there’s evidence that it could damage your gut, but because its structural
properties make it likely to produce unpleasant gut symptoms. Unlike carrageenan,
there’s no evidence that xanthan gum can cause serious harm (even in human studies
using doses much higher than people would normally encounter), so if you are able to
tolerate it, I see no compelling reason to strictly avoid it.
I wouldn’t recommend consuming large amounts every day, because xanthan gum
appears to have a high propensity for altering the gut microbiome, and it’s unclear
whether that alteration could be problematic in the long run. But the small amounts that
you would normally encounter in the context of an overall real-food diet shouldn’t
present a problem.
Guar Gum
I talked briefly about guar gum a while ago in my unexpectedly controversial article on
coconut milk, but I’ll give you a bit more detail here. Unlike xanthan gum, which is a
product of bacterial fermentation, guar gum is derived from an actual food: the guar
bean, or Indian cluster bean, which grows primarily in India and Pakistan. They look
similar to green beans, and are a common vegetable dish in the areas in which they
grow.
The physiological effects of guar gum have been extensively studied, first on animals and
then on humans. In rats, the only significant effects from guar gum supplementation were
reduced body weight and lower blood glucose, even with guar gum making up 15% of
the diet (over 100 times the FDA Acceptable Daily Intake). (55) Because guar gum is a
soluble fiber, neither of these effects is particularly surprising. Other animal studies
conducted to test the safety of guar gum concluded that it is not carcinogenic or
teratogenic (harmful to growing fetuses). (56, 57, 58)
Because the animal studies showed no harm even at very high doses, guar gum is now
being studied in humans as a therapeutic tool for reducing blood glucose and cholesterol
levels. Studies have shown guar gum supplementation to be effective for reducing
fasting blood glucose, improving glycemic control, reducing insulin requirements in
insulin-dependent diabetics, and reducing LDL cholesterol, although whether these
effects could be maintained long-term is uncertain. (59, 60, 61, 62, 63, 64)
Unfortunately, these studies do report gastrointestinal side effects such as increased gas.
In one study where subjects were given 21g of guar gum per day for 3 months, two
participants dropped out due to excessive gas and abdominal discomfort. (65)
Although 21g per day is far more guar gum than anyone would reasonably encounter in
their diet, even small amounts could cause unpleasant symptoms in those with sensitive
digestive systems, and I’ve had patients with gut issues improve after removing guar gum
from their diet. With that in mind, I think it makes sense to avoid guar gum if you have gut
issues, like small intestinal bacterial overgrowth (SIBO) or IBS, unless you’ve removed it
and added it back in without noticing any harmful effects.

Locust Bean Gum
Locust bean gum, also known as carob bean gum, is derived from the seeds of the carob
tree. During a two-year animal study, rats were given locust bean gum as 5% of their diet,
and no carcinogenic or other toxic effects were observed. (66)
Similar to guar gum, locust bean gum has also been studied in humans as a potential
cholesterol-lowering compound. (67) Normal subjects and subjects with familial
hypercholesterolemia were given between 8 and 30 grams per day of locust bean gum
for 8 weeks, resulting in reduced total cholesterol and an improved HDL to LDL ratio.
Participants did report increased gas, but it went away after a week or two, and no other
harmful effects were reported.
I think the same recommendation I gave for guar gum applies here: if you have gut
issues, it would probably be best to avoid locust bean gum. Otherwise, I see no
indication that it will cause harm.
Gum Arabic
Gum arabic is derived from the sap of the acacia tree. Under FDA regulations, gum arabic
is given an Acceptable Daily Intake level of ‘not specified,’ which is assigned to additives
with little or no observed toxic potential. Animal studies have shown that it is not
carcinogenic, mutagenic, or teratogenic, and even at very high doses, the animals did not
display any effects of toxicity. (68, 69)
In a small human study, 5 healthy men were given 25g of gum arabic per day for three
weeks, and no side effects were reported. (70) In fact, gum arabic had very little effect on
the participants, positive or negative, aside from a modest reduction in serum cholesterol
and an increase in breath hydrogen.
The increased breath hydrogen indicates metabolism by intestinal bacteria, which has
been confirmed by more recent studies on the prebiotic properties of gum arabic. A
study using healthy human volunteers found that gum arabic acts as a powerful prebiotic,
selectively stimulating the growth of bifidobacteria and lactobacilli. (71) The study authors
concluded that gum arabic is at least as effective a prebiotic as inulin, if not more so.
Many of you are probably aware that inulin is sold as a prebiotic supplement, so that’s
pretty significant!
Based on the available research, gum arabic seems pretty benign, even for those with
gut issues. I certainly wouldn’t be concerned about consuming small amounts of it,
although as always, be aware of your individual tolerance.
Tara Gum
Like guar gum and locust bean gum, tara gum is derived from the endosperm of a
legume. Tara gum is a relatively new food additive so there’s less data on it, but it has
been thoroughly studied for toxic effects in animals. Researchers conducted multiple 90-
day trials in rats, mice, and beagles with tara gum as 5% of the diet, and found no
adverse effects other than decreased body weight in the experimental groups. (72)
Three-generation reproductive rat studies and genotoxicity studies found no harmful
effects of tara gum. (73) In 2-year trials, the experimental groups had more tumors than
the control groups, but due to the “high spontaneous incidence” of this particular tumor
and the fact that nearly all of the control mice developed the tumor as well, researchers
concluded that this was not a result of the tara gum supplementation. (74)
I’m slightly more skeptical of tara gum compared with the other gums because the
toxicity results are less conclusive. Also, while all of the other gums have been tested on
humans, tara gum has not. That doesn’t mean it’s not safe, because the available
evidence indicates it is; it just means we don’t have as much to go on, and it’s always
good to be cautious of new food additives.
Gellan Gum
Gellan gum is similar to xanthan gum in that it is an exopolysaccharide produced by
bacterial fermentation. Unfortunately, the routine animal toxicity studies conducted for
new food additives aren’t available online, but we do have a human study to look at. To
test the safety of gellan gum, the diets of ten volunteers were supplemented with gellan
gum at approximately 30 times the level of normal dietary exposure for 23 days. (75)
Gellan gum acted as a bulking agent similar to xanthan gum, but no adverse effects were
reported. However, a rat study with gellan gum supplemented at 5% of the diet for 4
weeks resulted in abnormalities in intestinal microvilli, which is concerning. (76)
This rat study, as well as the lack of data overall, makes me cautious, and I think those
with sensitive guts should avoid it just to be on the safe side. For everyone else, I doubt
the small amounts found in food will cause a problem, but it might be best to avoid it if
possible.
Conclusion
As a general rule, gums can be problematic for those with digestive issues simply
because they’re mostly indigestible, but it’s very unlikely any of them will actually cause
harm. Of course it’s ideal to avoid food additives altogether, but I know it’s not realistic
for everyone to prepare all food from scratch, and unless you have digestive issues or a
sensitivity to certain gums, I don’t think it’s necessary.

Because I’ve covered a lot of different additives with similar applications (primarily
thickening or emulsifying), I’ll try to rank them for you. Let’s say you’re buying some
almond milk, and there are a bunch of different brands that use different additives. First,
do your best to avoid carrageenan. In general the concerns have been largely
overblown, but it definitely shows the highest potential for harm among the additives
we’ve discussed, and with all of the choices available to us, it should be pretty easy to
find a brand that doesn’t use it.
Next, I would avoid tara and gellan gums, not because they appear to be harmful, but
because we have less information on them.
I’d probably rank guar gum above xanthan gum because it’s derived from a food instead
of a bacterial exopolysaccharide, and it isn’t produced using common food allergens.
Locust bean gum is probably on about the same level as guar gum, although based on
the available studies, the gut symptoms associated with locust bean gum appear to be
less severe.
Gum arabic seems the least likely to create digestive symptoms, and it even stimulates
the growth of beneficial bacteria, so out of all the gums, it appears to be the least
problematic.
Finally, just remember that the overall quality of your diet is far more important than how
well you avoid these additives. Luckily, the two correlate pretty well!
chriskresser.com

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MAXIM Magazine Dr. Stephen Kroth 

What is GAINSWave, and why it is so effective for treating erectile dysfunction?

GAINSWave is a tested and documented protocol utilizing acoustically generated pressure waves, which enter the body at a prescribed intensity, frequency and depth. These waves create a type of “healthy inflammation” which triggers a rejuvenation response in the tissue treated.

With regular treatments over a short timeframe, this generates healthier and more responsive cells in erectile tissue. One of the earlier results is dissolving the micro plaque in vessels allowing for more blood flow and spiking the body’s natural nitric oxide levels. The long-term results are greater volume of erectile tissue and vascularization which is the increase in number of blood vessels.

Why would someone get this treatment instead of taking Cialis, Viagra or other ED prescription medication?

GAINSWave is a safe and powerful alternative to prescription medication. It also enhances the effects of prescribed meds (making them more effective or enabling patients to take smaller doses). However, the latter is only recommended in extreme circumstances.  Many men experience negative side effects with medication and do not like the limitations they present. Men should seriously consider utilizing GAINSWave if they are looking to treat the root cause of erectile dysfunction instead of just masking the symptoms. In addition to being one of the most beneficial solutions to Peyronie’s disease, men can also benefit from GAINSWave by taking a preventative approach to sexual wellness.

The body is a unique machine that needs maintenance, so, like changing the plugs, wires, and hoses on your muscle car, GAINSWave treatment is rejuvenating for tissues and vessels that create and maintain an erection.

What are the pros and cons of GAINSWave vs. ED medication?
No medication grows blood vessels, but the GAINSWave procedure does. The treatment also naturally spikes nitric oxide levels in the genitals, similar to a localized long-term Viagra response without the headache, facial flushing, nausea and tolerance to added chemicals in the body.  Men with scar tissue, regardless of even being aware of it yet, can see marked reduction in this thickening, banding, and curving that can choke off blood supply and create “dead zones” of sensitivity. The wave can reach the prostate through the pelvic floor during treatment, which has only positive effects on this most critical organ for male sexual enjoyment and male sexual health.

Many men report improved orgasm intensity and length as a result. The expansion of girth seen with treatment adds to the base strength of an erection, giving it a foundation for penetrative sex. Sensitivity improves as does refractory period (more frequent erections) and spontaneous erections.

The wave realigns the cells of connective tissue, promotes collagen growth and reduces pain and inflammation. This can result in previously retracted erectile tissue extending into more of the outer penis, ready for penetration and sensation. Some men report a normalizing of sensitivity, which means greater control over ejaculation timing and ability to sustain an erection in a condom.

There are no cons. Everyone sees something from a course of treatment. Regardless of a patient’s goals for reducing or eliminating medication, the treatment offers healing of the tissues that is just not possible with medication alone. Medication does not heal or improve the tissues – GAINSWave does.

How long does a typical GAINSWave treatment take and what happens during each appointment?

A standard treatment lasts about 20 minutes, a customized treatment for scar tissue slightly longer. The equipment we use at Enhanced Wellness Living is an FDA Cleared class I, non-abrasive medical device from Storz of Sweden, which produces an acoustic wave mechanically.  By way of a hand-held wand, this wave is transmitted via ultrasound gel into the tissue for a set number of pulses at determined protocol settings that allow for customization. Thus, the selection, training and experience level of the technician plays an important role. Areas of treatment include the perineal triangle, root of the penis, entire shaft, glands, frenulum and lower abdomen at the base of the penis.

What does a patient feel during treatments? And are there any side effects or recovery time needed?

Because this treatment is non-invasive there is zero downtime, and patients are encouraged to increase their sexual activity that day and throughout the course of treatment. Every patient receives topical numbing for the first treatment, mainly for the purpose of comfort.  Patients can feel the wave enter, can hear the machine and pulses, and describe a sensation of feeling the tissue transformed.  As far as negative side effects of GAINSWave, there are none.

How many treatments do most patients need?

The best results are seen between 6 to 12 treatments. Based on a patient’s age, health, medications and erectile-related conditions, maintenance treatments can vary from sporadic and rare to regular or on-demand.  The environmental and health factors that led to the development of E.D. can persist, so just like obtaining a fitness level, it requires maintenance based on the individual’s personal standards. The treatment is safe and free from harm, so there is no such thing as too many treatments.

A recent study in the scientific journal European Eurology showed that GAINSWave increased erection hardness and duration in all 20 of the patients tested. What has been the overall feedback of the patents you have treated?

Everyone sees positive results, and not just related to strength and rigidity of erection. Improvement in orgasm, sensation, erection frequency, prostate health, pain reduction, growth of blood vessels, reduction of scar tissue, increased girth…all of these are real and attainable by average people.

Additionally, patients have been seen to take the opportunity to improve communication with partners, expand their sexual expression, and notice tremendous empowerment in all areas of their life. Men do not seek this treatment to masturbate better, they seek the treatment to feel empowered and act on it, and to connect intimately with a partner more frequently in meaningful and fun ways.

Under what circumstances would you recommend GAINSWave to men experiencing ED?
Men with or without erectile problems will benefit from the therapy. I recommend men to take a preventive approach early on to nip any unwanted conditions to occur in the future while boosting their current performance. For those that are dealing with ED or Peyronie’s disease already, I strongly recommend they contact a GAINSWave provider to discuss treatment options.  Any male who does not have active cancer is a candidate.

Overall, how does GAINSWave rate as a treatment for men suffering from ED?

GAINSWave works at treating the root problem and promoting revitalized tissue. It’s not a “band-aid” just masking the symptoms, like medications.

Also, it removes the undesirable effect of creating more symptoms requiring additional medications. GAINSWave is changing the tissue and turning back the clock. Medication cannot do what the GAINSWave procedure does.

Full Article: https://hubs.ly/H0tk05q0

Enhanced Wellness Living, Ridgeland, MS, is proud to offer an array of sexual wellness treatment options for both men and women!

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Enhanced Wellness Living of Ridgeland, Mississippi is your leading Functional Medicine, Holistic Health, Lifestyle Medicine, and Integrative Medicine clinic!  Nurse Practitioner, Kelly Engelamann, and the Enhanced Wellness Living team have supported all of Mississippi including Ridgeland, Jackson, Madison, Brandon, Hernando, Starkville, Flowood, Vicksburg, Oxford, Olive Branch, the Mississippi Gulf, Cleveland, Clindon, Hattiesburg, Canton, Bolton, Hinds, and more since 2004!

These statements are not intended to diagnose, treat, or cure any disease.